I agree that this site is using cookies. You can find further informations
here
.
X
Login
Merkliste (
0
)
Home
About us
Home About us
Our history
Profile
Press & public relations
Friends
The library in figures
Exhibitions
Projects
Training, internships, careers
Films
Services & Information
Home Services & Information
Lending and interlibrary loans
Returns and renewals
Training and library tours
My Account
Library cards
New to the library?
Download Information
Opening hours
Learning spaces
PC, WLAN, copy, scan and print
Catalogs and collections
Home Catalogs and Collections
Rare books and manuscripts
Digital collections
Subject Areas
Our sites
Home Our sites
Central Library
Law Library (Juridicum)
BB Business and Economics (BB11)
BB Physics and Electrical Engineering
TB Engineering and Social Sciences
TB Economics and Nautical Sciences
TB Music
TB Art & Design
TB Bremerhaven
Contact the library
Home Contact the library
Staff Directory
Open access & publishing
Home Open access & publishing
Reference management: Citavi & RefWorks
Publishing documents
Open Access in Bremen
zur Desktop-Version
Toggle navigation
Merkliste
1 Ergebnisse
1
A composite origin of APOBEC3 signature mutations in breast..:
Michael A. Carpenter
;
Nuri A. Temiz
;
Mahmoud A. Ibrahim
...
doi:10.1371/journal.pgen.1011043.g005. , 2023
Link:
https://doi.org/10.1371/journal.pgen.1011043.g005
RT Journal T1
A composite origin of APOBEC3 signature mutations in breast cancer
UL https://suche.suub.uni-bremen.de/peid=base-ftdeakinunifig:oai:figshare.com:article_24696802&Exemplar=1&LAN=DE A1 Michael A. Carpenter A1 Nuri A. Temiz A1 Mahmoud A. Ibrahim A1 Matthew C. Jarvis A1 Margaret R. Brown A1 Prokopios P. Argyris A1 William L. Brown A1 Gabriel J. Starrett A1 Douglas Yee A1 Reuben S. Harris YR 2023 K1 Biophysics K1 Biochemistry K1 Medicine K1 Microbiology K1 Genetics K1 Molecular Biology K1 Immunology K1 Cancer K1 Infectious Diseases K1 Virology K1 Biological Sciences not elsewhere classified K1 whole genome sequences K1 triggers increased frequencies K1 stranded loop regions K1 standing etiologic debate K1 local sequence preferences K1 independent clones enabled K1 human cell line K1 catalytic mutant controls K1 70 % K1 suggesting K1 thymidine kinase </ K1 quantify genome mutation K1 overall mutation landscape K1 cellular apobec3 enzymes K1 div >< p K1 apobec3 signature mutations K1 although multiple enzymes K1 tk </ K1 g mutations K1 enzymes contribute K1 ytcw motifs K1 vast majority K1 therapeutic efforts K1 tct motifs K1 studies combine K1 similar tc K1 reports conflict K1 reporter gene K1 mutational impact K1 mutagenic activities K1 help resolve K1 future diagnostic K1 elevated rates K1 dna hairpins K1 confers sensitivity K1 combinatorial manner K1 >) gene JF doi:10.1371/journal.pgen.1011043.g005 LK http://dx.doi.org/https://doi.org/10.1371/journal.pgen.1011043.g005 DO https://doi.org/10.1371/journal.pgen.1011043.g005 SF ELIB - SuUB Bremen
Export
RefWorks (nur Desktop-Version!)
Flow
(Zuerst in
Flow
einloggen, dann importieren)