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1 Ergebnisse
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ALK G1269A mutation as a potential mechanism of acquired re..:
Sebastian Y. F. Michels
;
Andreas H. Scheel
;
Thomas Wündisch
...
https://doi.org/10.1038/s41698-017-0004-3. , 2017
Link:
https://doi.org/10.1038/s41698-017-0004-3
RT Journal T1
ALK G1269A mutation as a potential mechanism of acquired resistance to crizotinib in an ALK-rearranged inflammatory myofibroblastic tumor
UL https://suche.suub.uni-bremen.de/peid=base-ftdoajarticles:oai:doaj.org_article:33a80232e38044a3a05d9a9bbd74b52a&Exemplar=1&LAN=DE A1 Sebastian Y. F. Michels A1 Andreas H. Scheel A1 Thomas Wündisch A1 Johannes M. Heuckmann A1 Roopika Menon A1 Michael Puesken A1 Carsten Kobe A1 Helen Pasternack A1 Carina Heydt A1 Matthias Scheffler A1 Rieke Fischer A1 Anne M. Schultheis A1 Sabine Merkelbach-Bruse A1 Lukas Heukamp A1 Reinhard Büttner A1 Jürgen Wolf PB Nature Portfolio YR 2017 K1 Neoplasms. Tumors. Oncology. Including cancer and carcinogens K1 RC254-282 JF https://doi.org/10.1038/s41698-017-0004-3 LK http://dx.doi.org/https://doi.org/10.1038/s41698-017-0004-3 DO https://doi.org/10.1038/s41698-017-0004-3 SF ELIB - SuUB Bremen
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