I agree that this site is using cookies. You can find further informations
here
.
X
Login
Merkliste (
0
)
Home
About us
Home About us
Our history
Profile
Press & public relations
Friends
The library in figures
Exhibitions
Projects
Training, internships, careers
Films
Services & Information
Home Services & Information
Lending and interlibrary loans
Returns and renewals
Training and library tours
My Account
Library cards
New to the library?
Download Information
Opening hours
Learning spaces
PC, WLAN, copy, scan and print
Catalogs and collections
Home Catalogs and Collections
Rare books and manuscripts
Digital collections
Subject Areas
Our sites
Home Our sites
Central Library
Law Library (Juridicum)
BB Business and Economics (BB11)
BB Physics and Electrical Engineering
TB Engineering and Social Sciences
TB Economics and Nautical Sciences
TB Music
TB Art & Design
TB Bremerhaven
Contact the library
Home Contact the library
Staff Directory
Open access & publishing
Home Open access & publishing
Reference management: Citavi & RefWorks
Publishing documents
Open Access in Bremen
zur Desktop-Version
Toggle navigation
Merkliste
1 Ergebnisse
1
Hearts deficient in both Mfn1 and Mfn2 are protected agains..:
Hall, A.R
;
Burke, N
;
Dongworth, R.K
...
Hall, A.R, Burke, N, Dongworth, R.K, Kalkhoran, S.B, Dyson, A, Vicencio, J.M, Dorn, G.W, Yellon, D.M, Hausenloy, D.J (2016). Hearts deficient in both Mfn1 and Mfn2 are protected against acute myocardial infarction. Cell Death and Disease 7 (5) : e2238. ScholarBank@NUS Repository. https://doi.org/10.1038/cddis.2016.139. , 2016
Link:
https://scholarbank.nus.edu.sg/handle/10635/180381
RT Journal T1
Hearts deficient in both Mfn1 and Mfn2 are protected against acute myocardial infarction
UL https://suche.suub.uni-bremen.de/peid=base-ftnunivsingapore:oai:scholarbank.nus.edu.sg:10635_180381&Exemplar=1&LAN=DE A1 Hall, A.R A1 Burke, N A1 Dongworth, R.K A1 Kalkhoran, S.B A1 Dyson, A A1 Vicencio, J.M A1 Dorn, G.W A1 Yellon, D.M A1 Hausenloy, D.J PB Nature Publishing Group YR 2016 K1 antimycin A1 K1 calcium ion K1 cytochrome c oxidase K1 mitofusin 1 K1 mitofusin 2 K1 reactive oxygen metabolite K1 reduced nicotinamide adenine dinucleotide dehydrogenase (ubiquinone) K1 succinate dehydrogenase (ubiquinone) K1 ubiquinol cytochrome c reductase K1 calcium K1 carrier protein K1 guanosine triphosphatase K1 Mfn1 protein K1 mouse K1 Mfn2 protein K1 mitochondrial permeability transition pore K1 acute heart infarction K1 animal experiment K1 animal model K1 Article K1 cell organelle K1 confocal microscopy K1 controlled study K1 disorders of mitochondrial functions K1 echocardiography K1 fractional shortening K1 heart infarction size K1 heart left ventricle hypertrophy K1 heart mitochondrion K1 heart output K1 heart protection K1 mitochondrion swelling K1 myocardial ischemia reperfusion injury K1 nonhuman K1 priority journal K1 protein expression K1 animal K1 cardiac muscle K1 cardiac muscle cell K1 deficiency K1 drug effects K1 gene expression K1 genetics K1 knockout mouse K1 metabolism K1 mitochondrial dynamics K1 mitochondrion JF Hall, A.R, Burke, N, Dongworth, R.K, Kalkhoran, S.B, Dyson, A, Vicencio, J.M, Dorn, G.W, Yellon, D.M, Hausenloy, D.J (2016). Hearts deficient in both Mfn1 and Mfn2 are protected against acute myocardial infarction. Cell Death and Disease 7 (5) : e2238. ScholarBank@NUS Repository. https://doi.org/10.1038/cddis.2016.139 LK http://dx.doi.org/https://scholarbank.nus.edu.sg/handle/10635/180381 DO https://scholarbank.nus.edu.sg/handle/10635/180381 SF ELIB - SuUB Bremen
Export
RefWorks (nur Desktop-Version!)
Flow
(Zuerst in
Flow
einloggen, dann importieren)